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RcsA-dependent and -independent growth defects caused by the activated Rcs phosphorelay system in the Escherichia coli pgsA null mutant
https://sucra.repo.nii.ac.jp/records/12608
https://sucra.repo.nii.ac.jp/records/126082c6efc54-953d-45aa-afb8-de686926c9e5
名前 / ファイル | ライセンス | アクション |
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A1002020.pdf (1.5 MB)
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Item type | 学術雑誌論文 / Journal Article(1) | |||||
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公開日 | 2007-03-26 | |||||
タイトル | ||||||
タイトル | RcsA-dependent and -independent growth defects caused by the activated Rcs phosphorelay system in the Escherichia coli pgsA null mutant | |||||
言語 | ||||||
言語 | eng | |||||
資源タイプ | ||||||
資源タイプ識別子 | http://purl.org/coar/resource_type/c_6501 | |||||
資源タイプ | journal article | |||||
著者 |
原, 弘志
× 原, 弘志 |
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著者 ローマ字 | ||||||
Hara, Hiroshi | ||||||
著者 ローマ字 | ||||||
Nagahama, Hideki | ||||||
著者 ローマ字 | ||||||
Sakamoto, Yutaka | ||||||
著者 ローマ字 | ||||||
Matsumoto, Kouji | ||||||
著者 所属 | ||||||
埼玉大学大学院理工学研究科 | ||||||
著者 所属(別言語) | ||||||
Graduate School of Science and Engineering, Saitama University | ||||||
書誌情報 |
JOURNAL OF GENERAL AND APPLIED MICROBIOLOGY 巻 52, 号 2, 発行日 2006 |
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年月次 | ||||||
2006 | ||||||
出版者名 | ||||||
出版者 | 財団法人 応用微生物学研究奨励会 | |||||
DOI | ||||||
識別子タイプ | DOI | |||||
関連識別子 | info:doi/10.2323/jgam.52.91 | |||||
抄録 | ||||||
内容記述タイプ | Abstract | |||||
内容記述 | In the Escherichia coli pgsA null mutant, which lacks the major acidic phospholipids, theRcs phosphorelay signal transduction system is activated, causing thermosensitivegrowth. The mutant grows poorly at 37°C and lyses at 42°C. We showed that the poorgrowth at 37°C was corrected by disruption of the rcsA gene, which codes for acoregulator protein that interacts with the RcsB response regulator of the phosphorelay system. However, mutant cells still lysed when incubated at 42°C evenin the absence of RcsA. We conclude that the activated Rcs phosphorelay in the pgsAnull mutant has both RcsA-dependent and -independent growth inhibitory effects.Since the Rcs system has been shown to positively regulate the essential cell divisiongenes ftsA and ftsZ independently of RcsA, we measured cellular levels of the FtsZ protein, but found that the growth defect of the mutant at 42°C did not involve achange in the level of this protein. | |||||
資源タイプ | ||||||
内容記述タイプ | Other | |||||
内容記述 | text | |||||
フォーマット | ||||||
内容記述タイプ | Other | |||||
内容記述 | application/pdf | |||||
作成日 | ||||||
日付 | 2007-03-05 | |||||
日付タイプ | Created | |||||
アイテムID | ||||||
A1002020 |