@article{oai:sucra.repo.nii.ac.jp:00016518,
 author = {田中, 秀逸 and 井上, 弘一 and 畠山, 晋},
 journal = {埼玉大学地域オープンイノベーションセンター紀要, Report of Comprehensive Open Innovation Center, Saitama University},
 month = {},
 note = {At gene targeting in Neuroapora, the deficient strains of non-homologous end joining (NHEJ), a mechanism for repair of DNA double strand break, induce amazing high efficiency. We had found this technique and gotten a patent concerning it. We had already checked disrupted strains of genes encoding XRCC
and XLF homologes, adding to KU70, KU80, or LIG4 homologues, which have directly roles in the NHEJ pathway. In this study, we have newly checked disrupted strains of genes mus-23, uvs-6, mus-45, which encode each protein for MRX protein trimer, respectively. It is known that MRX complex works on upstream of the NHEJ pathway in DNA repair. We also examine characters of random insertion of DNA fragments
injected into cells in Neurospora., text, application/pdf},
 pages = {21--22},
 title = {糸状菌における組換えに関する研究},
 volume = {2},
 year = {2010},
 yomi = {タナカ, シュウイツ and イノウエ, ヒロカズ and ハタケヤマ, シン}
}